Preliminary experiments indicate conolidine may inhibit distinct ion channels, lowering neuronal excitability and limiting ache indicators. This system is particularly suitable in neuropathic pain, in which abnormal signaling triggers persistent pain. Additionally, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to pain notion. Computational docking scie... https://fanb062pix6.bloggazzo.com/profile